Neuropsychic pathology in the context of anterior
Neuropsychic pathology in the context of anterior

Summary/Abstract: Acute pain is differentiated from chronic pain according to the parameters analyzed by the International Association for the Study of Pain (IASP): duration, etiology, mechanism, character, mental state of the patient. The substrate of pain in somatic and visceral damage is the release of substances with a double neuromediating and vasoactive effect, which act indirectly on the membrane of nociceptors through local biochemical changes or directly, through mechanical or chemical action. Microcirculation changes either through vasoconstriction, influencing capillary permeability and determining the level of algogenic substances (serotonin, histamine and bradykinin) and stimulating the nociceptive reaction, or through vasodilatation, with different mechanisms and pathogenesis.The damage to the peripheral nerves and the inhibitory influences known in patients with diabetes or in post herpetic neuralgia, influence the synaptic transmission at the level of cells in the posterior horns, which could explain the persistence of pain and therefore the modification of one or more defining parameters (duration, intensity, character) or likewise, psycho-emotional stress, which releasing a series of algogenic substances can induce spasm of the skeletal muscles, respectively local vasoconstriction, ultimately causing visceral and somatic dysfunction at the same time, of which neuropsychiatric damage remains an extremely topical problem.

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